Bøger af Kiran Bala

This book discusses the current state of strategies that utilize the ability of microbes to remediate waste sources, such as wastewater streams and mine tails, and provide environmentally friendly options to mitigate soil and water pollution caused due to heavy metals. It also provides details about the development of biodegradable plastics from microbial sources and how they can be economical and greener alternatives to the currently used options. It will act as a single platform for combining the remedial powers of microbes which can be both sustainable and practical under a single volume. This text will be particularly useful for govt. institutions, academicians, and industry professionals, who deal in wastewater remediation and sustainability of currently used sources of plastics and other high-value metabolites. It will also be of practical help to engineers involved in remediation processes for wastewater and industrial waste.

T2DM is a leading disease in the human people throughout the world, mainly in developing countries with an incidence of 9.3% (Buse et al, 2020). It is characterized by a chronically elevated high blood glucose level due to impairment in insulin action, insulin secretion or both and dysregulation of protein and lipid metabolism. Other factor responsible for pathogenesis of T2DM are combined action of environmental, dietary, behavioral and genetic risk factors which play a major role in describing the susceptibility causes of type 2 diabetes mellitus (Gloyn et al, 2018).T2DM is mainly linked with obesity, age, overweight, family history, hypertension, urbanization and oxidative stress (Sharma et al, 2019). T2DM is a complex polygenic and heterogeneous disorder because it depends on multiple genetic loci and environmental factors (Apovian et al, 2016). Environmental factors can lead to diabetes by affecting gene expression through epigenetic modifications. Around 30%-70% of T2DM risk can be recognized to inherited factors (Cariaso et al, 2012). GWAS identified several genetic loci linked with T2DM (Florez et al, 2008).